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Acute Pancreatitis-Induced Takotsubo Cardiomyopathy in an African American Male.

Bruenjes JD, Vallabhajosyula S, Vacek CJ, Fixley JE - ACG Case Rep J (2015)

Bottom Line: Takotsubo cardiomyopathy (TCM) is triggered by multiple physical and psychological stressors and frequently mimics acute coronary syndrome.Laboratory parameters revealed elevated troponin-I, amylase, lipase, and metabolic acidosis.Coronary angiogram was unrevealing for coronary atherosclerosis and he was managed conservatively for acute pancreatitis and heart failure from TCM.

View Article: PubMed Central - PubMed

Affiliation: Creighton University School of Medicine, Omaha, NE.

ABSTRACT
Takotsubo cardiomyopathy (TCM) is triggered by multiple physical and psychological stressors and frequently mimics acute coronary syndrome. Acute pancreatitis as a trigger for TCM has rarely been reported. We report a 55-year-old African American man with hypertension and alcohol abuse history, who presented with epigastric and sub-sternal pain and electrocardiogram demonstrating ischemic changes. Laboratory parameters revealed elevated troponin-I, amylase, lipase, and metabolic acidosis. He was diagnosed with acute pancreatitis and ACS. Coronary angiogram was unrevealing for coronary atherosclerosis and he was managed conservatively for acute pancreatitis and heart failure from TCM.

No MeSH data available.


Related in: MedlinePlus

Transthoracic echocardiogram 3 weeks after discharge demonstrating normal LV ejection fraction with no wall motion abnormalities.
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Figure 5: Transthoracic echocardiogram 3 weeks after discharge demonstrating normal LV ejection fraction with no wall motion abnormalities.

Mentions: Over the next 2-3 days, his pancreatitis improved and he was able to tolerate an oral diet with minimal pain and regular bowel movements. EKG at discharge showed normalization of ST-T segment depression and improving T-wave inversions (Figure 4). A repeat echocardiogram 3 weeks later demonstrated complete normalization of LV function highlighting the transient nature of this disease, essential for diagnosis (Figure 5).5 His anticoagulation was discontinued and he continues to be asymptomatic on follow-up.


Acute Pancreatitis-Induced Takotsubo Cardiomyopathy in an African American Male.

Bruenjes JD, Vallabhajosyula S, Vacek CJ, Fixley JE - ACG Case Rep J (2015)

Transthoracic echocardiogram 3 weeks after discharge demonstrating normal LV ejection fraction with no wall motion abnormalities.
© Copyright Policy - open-access
Related In: Results  -  Collection

License
Show All Figures
getmorefigures.php?uid=PMC4612760&req=5

Figure 5: Transthoracic echocardiogram 3 weeks after discharge demonstrating normal LV ejection fraction with no wall motion abnormalities.
Mentions: Over the next 2-3 days, his pancreatitis improved and he was able to tolerate an oral diet with minimal pain and regular bowel movements. EKG at discharge showed normalization of ST-T segment depression and improving T-wave inversions (Figure 4). A repeat echocardiogram 3 weeks later demonstrated complete normalization of LV function highlighting the transient nature of this disease, essential for diagnosis (Figure 5).5 His anticoagulation was discontinued and he continues to be asymptomatic on follow-up.

Bottom Line: Takotsubo cardiomyopathy (TCM) is triggered by multiple physical and psychological stressors and frequently mimics acute coronary syndrome.Laboratory parameters revealed elevated troponin-I, amylase, lipase, and metabolic acidosis.Coronary angiogram was unrevealing for coronary atherosclerosis and he was managed conservatively for acute pancreatitis and heart failure from TCM.

View Article: PubMed Central - PubMed

Affiliation: Creighton University School of Medicine, Omaha, NE.

ABSTRACT
Takotsubo cardiomyopathy (TCM) is triggered by multiple physical and psychological stressors and frequently mimics acute coronary syndrome. Acute pancreatitis as a trigger for TCM has rarely been reported. We report a 55-year-old African American man with hypertension and alcohol abuse history, who presented with epigastric and sub-sternal pain and electrocardiogram demonstrating ischemic changes. Laboratory parameters revealed elevated troponin-I, amylase, lipase, and metabolic acidosis. He was diagnosed with acute pancreatitis and ACS. Coronary angiogram was unrevealing for coronary atherosclerosis and he was managed conservatively for acute pancreatitis and heart failure from TCM.

No MeSH data available.


Related in: MedlinePlus