Limits...
Masson ’ s tumour of the kidney

View Article: PubMed Central

ABSTRACT

In 1923 Masson described a neo-plastic process consisting of papillary hyperplasia of the vascular endothelial cells, with a consequent obliteration of the vascular lumen, followed by degenerative changes. He introduced the term ‘vegetant intravascular hemangioendothelioma’. However, these days it is more commonly known as papillary endothelial hyperplasia (PEH)[1]. Although relatively rare, there are numerous accounts of PEH in literature, describing its predilection for the head and neck region. Our case report describes the finding of a PEH within the kidney; a site previously described only once in the literature.

No MeSH data available.


Color-coded duplex sonography, coronal plane. The lesion that is slightly hyperechoic compared to the renal parenchyma and Doppler spectrum demonstrates intralesional flow with a normal resistance pattern (resistance index 0.69).
© Copyright Policy
Related In: Results  -  Collection


getmorefigures.php?uid=PMC4554702&req=5

Fig1: Color-coded duplex sonography, coronal plane. The lesion that is slightly hyperechoic compared to the renal parenchyma and Doppler spectrum demonstrates intralesional flow with a normal resistance pattern (resistance index 0.69).

Mentions: A 64-year-old man was admitted to the hospital for resection of a sigmoid colon adenocarcinoma. He had no medical antecedents. Physical examination was normal. There was no flank mass or tenderness, nor cutaneous vascular lesions. Routine laboratory tests were normal. Renal ultrasound demonstrated a small rounded lesion in the centre of the left kidney (Fig. 1). The lesion was slightly hyperechoic compared to the renal cortex. Color-coded duplex sonography demonstrated intralesional flow, with a resistance index of 0.69. Heterogeneous lesional enhancement was seen on contrast enhanced computed tomography (CT) (Fig. 2); the lesion was isodense on to the renal parenchyma on pre-contrast scans. MRI was performed, showing a lesion isointense to the renal cortex on T1-weighted images (TR 7.7/TE 4.2 msec) (Fig. 3) and predominantly hyperintense on T2-weighted images (TR 4.3/TE 60 msec) (Fig. 4(a,b)). The latter also demonstrated small intratumoral hypointense strands, fanning out from the medial border of the lesion. 3D contrast-enhanced MR-angiography (Fig. 5) in the coronal plane demonstrated lesional enhancement, slightly less than the renal cortex. The renal artery and vein appeared normal. The tumor abutted the renal pelvis without invasion. No metastases were demonstrated.Figure 1


Masson ’ s tumour of the kidney
Color-coded duplex sonography, coronal plane. The lesion that is slightly hyperechoic compared to the renal parenchyma and Doppler spectrum demonstrates intralesional flow with a normal resistance pattern (resistance index 0.69).
© Copyright Policy
Related In: Results  -  Collection

Show All Figures
getmorefigures.php?uid=PMC4554702&req=5

Fig1: Color-coded duplex sonography, coronal plane. The lesion that is slightly hyperechoic compared to the renal parenchyma and Doppler spectrum demonstrates intralesional flow with a normal resistance pattern (resistance index 0.69).
Mentions: A 64-year-old man was admitted to the hospital for resection of a sigmoid colon adenocarcinoma. He had no medical antecedents. Physical examination was normal. There was no flank mass or tenderness, nor cutaneous vascular lesions. Routine laboratory tests were normal. Renal ultrasound demonstrated a small rounded lesion in the centre of the left kidney (Fig. 1). The lesion was slightly hyperechoic compared to the renal cortex. Color-coded duplex sonography demonstrated intralesional flow, with a resistance index of 0.69. Heterogeneous lesional enhancement was seen on contrast enhanced computed tomography (CT) (Fig. 2); the lesion was isodense on to the renal parenchyma on pre-contrast scans. MRI was performed, showing a lesion isointense to the renal cortex on T1-weighted images (TR 7.7/TE 4.2 msec) (Fig. 3) and predominantly hyperintense on T2-weighted images (TR 4.3/TE 60 msec) (Fig. 4(a,b)). The latter also demonstrated small intratumoral hypointense strands, fanning out from the medial border of the lesion. 3D contrast-enhanced MR-angiography (Fig. 5) in the coronal plane demonstrated lesional enhancement, slightly less than the renal cortex. The renal artery and vein appeared normal. The tumor abutted the renal pelvis without invasion. No metastases were demonstrated.Figure 1

View Article: PubMed Central

ABSTRACT

In 1923 Masson described a neo-plastic process consisting of papillary hyperplasia of the vascular endothelial cells, with a consequent obliteration of the vascular lumen, followed by degenerative changes. He introduced the term ‘vegetant intravascular hemangioendothelioma’. However, these days it is more commonly known as papillary endothelial hyperplasia (PEH)[1]. Although relatively rare, there are numerous accounts of PEH in literature, describing its predilection for the head and neck region. Our case report describes the finding of a PEH within the kidney; a site previously described only once in the literature.

No MeSH data available.