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Correction: A role for Tn6029 in the evolution of the complex antibiotic resistance gene loci in genomic island 3 in enteroaggregative hemorrhagic Escherichia coli O104:H4.

- PLoS ONE (2015)

View Article: PubMed Central - PubMed

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Molecular signatures created by the insertion of IS26.A: The 3´-CS of class 1 integrons in pASL01a and pAKU_1 has been structurally modified by different IS26-mediated deletion events such that PCR with L1 and JL-D2 primers is expected to generate 2082 and 2342 bp long amplicons respectively. B: Eight base pair signature sequences created by the insertion of IS26 found flanking the inverted repeats of IS26 elements clearly suggest that the CRL in strain 2011EC-3493 is a derivative of Tn6029C while 2009–2050 is a derivative of Tn6029B described previously in pHCM1. The eight base repeat on the left end of Tn6029B and that in 2009EL-2050 are different because in pHCM1 (Tn6029B) there is a deletion of the class 1 integron whereas in 2009EL-2050 the fragment adjacent to the left hand end of the insertion site is inverted by IS26-mediated events. These signatures are also consistent with the proposition that Tn6029C evolved from Tn6029B.
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pone.0126197.g001: Molecular signatures created by the insertion of IS26.A: The 3´-CS of class 1 integrons in pASL01a and pAKU_1 has been structurally modified by different IS26-mediated deletion events such that PCR with L1 and JL-D2 primers is expected to generate 2082 and 2342 bp long amplicons respectively. B: Eight base pair signature sequences created by the insertion of IS26 found flanking the inverted repeats of IS26 elements clearly suggest that the CRL in strain 2011EC-3493 is a derivative of Tn6029C while 2009–2050 is a derivative of Tn6029B described previously in pHCM1. The eight base repeat on the left end of Tn6029B and that in 2009EL-2050 are different because in pHCM1 (Tn6029B) there is a deletion of the class 1 integron whereas in 2009EL-2050 the fragment adjacent to the left hand end of the insertion site is inverted by IS26-mediated events. These signatures are also consistent with the proposition that Tn6029C evolved from Tn6029B.

Mentions: The image for Fig 2 is incorrect. The publisher apologizes for the error. Please see the corrected Fig 2 here.


Correction: A role for Tn6029 in the evolution of the complex antibiotic resistance gene loci in genomic island 3 in enteroaggregative hemorrhagic Escherichia coli O104:H4.

- PLoS ONE (2015)

Molecular signatures created by the insertion of IS26.A: The 3´-CS of class 1 integrons in pASL01a and pAKU_1 has been structurally modified by different IS26-mediated deletion events such that PCR with L1 and JL-D2 primers is expected to generate 2082 and 2342 bp long amplicons respectively. B: Eight base pair signature sequences created by the insertion of IS26 found flanking the inverted repeats of IS26 elements clearly suggest that the CRL in strain 2011EC-3493 is a derivative of Tn6029C while 2009–2050 is a derivative of Tn6029B described previously in pHCM1. The eight base repeat on the left end of Tn6029B and that in 2009EL-2050 are different because in pHCM1 (Tn6029B) there is a deletion of the class 1 integron whereas in 2009EL-2050 the fragment adjacent to the left hand end of the insertion site is inverted by IS26-mediated events. These signatures are also consistent with the proposition that Tn6029C evolved from Tn6029B.
© Copyright Policy
Related In: Results  -  Collection

License
Show All Figures
getmorefigures.php?uid=PMC4401760&req=5

pone.0126197.g001: Molecular signatures created by the insertion of IS26.A: The 3´-CS of class 1 integrons in pASL01a and pAKU_1 has been structurally modified by different IS26-mediated deletion events such that PCR with L1 and JL-D2 primers is expected to generate 2082 and 2342 bp long amplicons respectively. B: Eight base pair signature sequences created by the insertion of IS26 found flanking the inverted repeats of IS26 elements clearly suggest that the CRL in strain 2011EC-3493 is a derivative of Tn6029C while 2009–2050 is a derivative of Tn6029B described previously in pHCM1. The eight base repeat on the left end of Tn6029B and that in 2009EL-2050 are different because in pHCM1 (Tn6029B) there is a deletion of the class 1 integron whereas in 2009EL-2050 the fragment adjacent to the left hand end of the insertion site is inverted by IS26-mediated events. These signatures are also consistent with the proposition that Tn6029C evolved from Tn6029B.
Mentions: The image for Fig 2 is incorrect. The publisher apologizes for the error. Please see the corrected Fig 2 here.

View Article: PubMed Central - PubMed

No MeSH data available.


Related in: MedlinePlus