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Spontaneous coronary artery dissection in polycystic kidney disease.

Klingenberg-Salachova F, Limburg S, Boereboom F - Clin Kidney J (2012)

Bottom Line: We suggest that the genetic disorder in ADPKD is the main cause of instable artery vasculature.Our case also shows that CAD can be missed in the acute phase.The patient received medical management.

View Article: PubMed Central - PubMed

Affiliation: Department of General Medicine, Diakonessenhuis, Utrecht, The Netherlands.

ABSTRACT
Little is known about the association between autosomal-dominant polycystic kidney disease (ADPKD) and coronary artery dissection (CAD). We suggest that the genetic disorder in ADPKD is the main cause of instable artery vasculature. Our case also shows that CAD can be missed in the acute phase. Therefore, we recommend additional investigation in patients with ADPKD presenting with acute chest pain. We report a case of a patient who developed a myocardial infarction due to a spontaneous dissection of the left anterior descending coronary artery. ADPKD was diagnosed during the additional investigation. The patient received medical management.

No MeSH data available.


Related in: MedlinePlus

The CAG (A) shows the compression of the LAD coronary artery during the systole resulting in narrowing and (B) performed after the second chest pain attack demonstrating a dissection in the distal left anterior descending coronary artery.
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fig2: The CAG (A) shows the compression of the LAD coronary artery during the systole resulting in narrowing and (B) performed after the second chest pain attack demonstrating a dissection in the distal left anterior descending coronary artery.

Mentions: Based on these results, a presumptive diagnosis of acute septal myocardial infarction was made. The patient was immediately transported to the University Medical Centre for cardiac intervention. Coronary artery angiography (CAG) revealed a transient occlusion of the left anterior descending (LAD) coronary artery, most probably as a result of myocardial bridging (Figure 2A). Other coronary arteries appeared normal. Percutaneous coronary intervention was not performed due to small vessel size. The myocardial infarction was treated with anti-platelet therapy, statin and metoprolol.


Spontaneous coronary artery dissection in polycystic kidney disease.

Klingenberg-Salachova F, Limburg S, Boereboom F - Clin Kidney J (2012)

The CAG (A) shows the compression of the LAD coronary artery during the systole resulting in narrowing and (B) performed after the second chest pain attack demonstrating a dissection in the distal left anterior descending coronary artery.
© Copyright Policy - creative-commons
Related In: Results  -  Collection

License
Show All Figures
getmorefigures.php?uid=PMC4400459&req=5

fig2: The CAG (A) shows the compression of the LAD coronary artery during the systole resulting in narrowing and (B) performed after the second chest pain attack demonstrating a dissection in the distal left anterior descending coronary artery.
Mentions: Based on these results, a presumptive diagnosis of acute septal myocardial infarction was made. The patient was immediately transported to the University Medical Centre for cardiac intervention. Coronary artery angiography (CAG) revealed a transient occlusion of the left anterior descending (LAD) coronary artery, most probably as a result of myocardial bridging (Figure 2A). Other coronary arteries appeared normal. Percutaneous coronary intervention was not performed due to small vessel size. The myocardial infarction was treated with anti-platelet therapy, statin and metoprolol.

Bottom Line: We suggest that the genetic disorder in ADPKD is the main cause of instable artery vasculature.Our case also shows that CAD can be missed in the acute phase.The patient received medical management.

View Article: PubMed Central - PubMed

Affiliation: Department of General Medicine, Diakonessenhuis, Utrecht, The Netherlands.

ABSTRACT
Little is known about the association between autosomal-dominant polycystic kidney disease (ADPKD) and coronary artery dissection (CAD). We suggest that the genetic disorder in ADPKD is the main cause of instable artery vasculature. Our case also shows that CAD can be missed in the acute phase. Therefore, we recommend additional investigation in patients with ADPKD presenting with acute chest pain. We report a case of a patient who developed a myocardial infarction due to a spontaneous dissection of the left anterior descending coronary artery. ADPKD was diagnosed during the additional investigation. The patient received medical management.

No MeSH data available.


Related in: MedlinePlus