Limits...
Mediastinal teratoma with hydrops fetalis in a newborn and development of chronic respiratory insufficiency.

Simoncic M, Kopriva S, Zupancic Z, Jerse M, Babnik J, Srpcic M, Grosek S - Radiol Oncol (2014)

Bottom Line: Early surgical removal is important, but, the result of treatment depends on the stage of development of mediastinal organs and complications in the postoperative period.Histology examination revealed an encapsulated immature teratoma.Additional diagnostic search revealed abnormal course of both pulmonary arteries, which was probably one of the main causes of respiratory insufficiency.

View Article: PubMed Central - PubMed

Affiliation: Department of Pediatric Surgery and Intensive Care, University Medical Centre Ljubljana, Ljubljana, Slovenia.

ABSTRACT

Background: Mediastinal fetal teratoma can be detected as a mass in the chest during a routine prenatal ultra-sound screening. Because of the pressure on mediastinal structures it can be the cause of non-immune hydrops fetalis and polyhydramnion. The development of hydrops fetalis leads to fetal death or premature delivery in most reported cases. Early surgical removal is important, but, the result of treatment depends on the stage of development of mediastinal organs and complications in the postoperative period.

Case report: A 31-year-old gravida carrying twins, with spontaneous membrane rupture at 32 weeks gestation underwent urgent caesarean section after antenatal ultrasound revealed severe polyhydramnion and hydrops fetalis in geminus A. The child was intubated immediately after birth due to severe respiratory distress. Ultrasound and X-ray revealed a tumour mass in the right hemithorax. Tumour resection was performed at the age of 7 days. Histology examination revealed an encapsulated immature teratoma. The postoperative course was complicated with respiratory insufficiency which turned into chronic at the age of eight months.

Conclusion: This is the fifth reported child with fetal mediastinal teratoma and severe hydrops fetalis that survived the neonatal period. Additional diagnostic search revealed abnormal course of both pulmonary arteries, which was probably one of the main causes of respiratory insufficiency.

No MeSH data available.


Related in: MedlinePlus

Geminus A with severe hydrops fetalis was intubated immediately after birth.
© Copyright Policy - open-access
Related In: Results  -  Collection

License 1 - License 2
getmorefigures.php?uid=PMC4230561&req=5

f1-rado-48-04-397: Geminus A with severe hydrops fetalis was intubated immediately after birth.

Mentions: A 31-year-old gravida 2 with twins, para 1, was referred to the University Medical Centre Ljubljana because of spontaneous membrane rupture at 32 weeks gestation after an uncomplicated pregnancy. After admission US revealed polyhydramnion and severe NIHF in geminus A. Due to life threatening condition also for healthy geminus B an urgent caesarean section was performed. Geminus A was a boy with Apgar score 3/5 and a birth weight of 3,200 g. He was intubated immediately after birth because of severe NIHF and respiratory distress (Figure 1).


Mediastinal teratoma with hydrops fetalis in a newborn and development of chronic respiratory insufficiency.

Simoncic M, Kopriva S, Zupancic Z, Jerse M, Babnik J, Srpcic M, Grosek S - Radiol Oncol (2014)

Geminus A with severe hydrops fetalis was intubated immediately after birth.
© Copyright Policy - open-access
Related In: Results  -  Collection

License 1 - License 2
Show All Figures
getmorefigures.php?uid=PMC4230561&req=5

f1-rado-48-04-397: Geminus A with severe hydrops fetalis was intubated immediately after birth.
Mentions: A 31-year-old gravida 2 with twins, para 1, was referred to the University Medical Centre Ljubljana because of spontaneous membrane rupture at 32 weeks gestation after an uncomplicated pregnancy. After admission US revealed polyhydramnion and severe NIHF in geminus A. Due to life threatening condition also for healthy geminus B an urgent caesarean section was performed. Geminus A was a boy with Apgar score 3/5 and a birth weight of 3,200 g. He was intubated immediately after birth because of severe NIHF and respiratory distress (Figure 1).

Bottom Line: Early surgical removal is important, but, the result of treatment depends on the stage of development of mediastinal organs and complications in the postoperative period.Histology examination revealed an encapsulated immature teratoma.Additional diagnostic search revealed abnormal course of both pulmonary arteries, which was probably one of the main causes of respiratory insufficiency.

View Article: PubMed Central - PubMed

Affiliation: Department of Pediatric Surgery and Intensive Care, University Medical Centre Ljubljana, Ljubljana, Slovenia.

ABSTRACT

Background: Mediastinal fetal teratoma can be detected as a mass in the chest during a routine prenatal ultra-sound screening. Because of the pressure on mediastinal structures it can be the cause of non-immune hydrops fetalis and polyhydramnion. The development of hydrops fetalis leads to fetal death or premature delivery in most reported cases. Early surgical removal is important, but, the result of treatment depends on the stage of development of mediastinal organs and complications in the postoperative period.

Case report: A 31-year-old gravida carrying twins, with spontaneous membrane rupture at 32 weeks gestation underwent urgent caesarean section after antenatal ultrasound revealed severe polyhydramnion and hydrops fetalis in geminus A. The child was intubated immediately after birth due to severe respiratory distress. Ultrasound and X-ray revealed a tumour mass in the right hemithorax. Tumour resection was performed at the age of 7 days. Histology examination revealed an encapsulated immature teratoma. The postoperative course was complicated with respiratory insufficiency which turned into chronic at the age of eight months.

Conclusion: This is the fifth reported child with fetal mediastinal teratoma and severe hydrops fetalis that survived the neonatal period. Additional diagnostic search revealed abnormal course of both pulmonary arteries, which was probably one of the main causes of respiratory insufficiency.

No MeSH data available.


Related in: MedlinePlus