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Mentions: Whole genome analysis using PCA followed by denoising revealed that principal component 22, which accounted for 0.5% of the total variance, was significantly associated with the treatment variable: [DE 6hr and 30hr] versus [DE 0hr, FA 0hr, 6hr, and 30hr] (Figure 1). Using loading cutoff of ±6 standard deviations, we found 89 CpG sites to possess the specific pattern of variation (Figure 2). These include genes whose expression is associated with exposure to either diesel exhaust or components of diesel exhaust as reported by literature: CASP7, ATCAY, ABCA1, JAK3, CYFIP2, and NOX2 [1-6].
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