Open-i Logo
Submit this form Query By Image

Internet Explorer requires you to use Upload Image button. Other browsers support the ability to drag and drop the image to anywhere in the browser window to perform an Image Search or use the Upload Image button.

Supported File Types are: .jpeg, .jpg, .gif and .png.
Results 1 - 20 of 408767 1 2 3 4 5 Next View as List View Image Grid View

- Selected Limits -

Query By Image

Similar Collection Image

Rank By

Image Type

Subsets

Specialties

Search In
Apg14p and Vps38p exist in distinct complexes. (A and B) AKY73 cells were grown in YPD at 30°C. Total lysates were solubilized with Triton X-100 and incubated with protein A–immobilized anti-Apg14p antibodies (lane 1) or anti-Vps38p antibodies (lane 2). The beads were then separated into two fractions and were subjected to immunoblotting (A) or PtdIns 3–kinase assays (B). In A, bound proteins were eluted from the beads, separated by SDS-PAGE, and detected by immunoblotting with anti-Vps30p, anti-Vps34p, anti-Vps15p, anti-Apg14p, and anti-Vps38p antibodies. In B, labeled lipids were extracted, separated by TLC, and visualized by autoradiography using BAS2000. (C) AKY112/pAUR112 (vector; lane 1) and AKY112/pKHR69 (His6–Myc–APG14; lane 2) cell lysates were solubilized with Triton X-100 and loaded on a Ni-NTA agarose column. Bound proteins were eluted with 250 mM imidazole and were subjected to immunoblotting with anti-Vps30p, anti-Myc (9E10), anti-Vps34p, and anti-Vps38p antibodies. Mean difference waveforms for adult data on the Circles (black) and the Rabbits (grey) paradigm. The time points where the stimulus appeared (Stimulus), part of the stimulus reappeared and the response cue (Response cue) was given, and the period of interest (POI) for calculation of the laterality index is also indicated. Ectopic expression of p16INK4a results in reduction of mkk7−/− BMMC hyperproliferation. Mkk7 deficient and wild-type BMMCs were infected with empty virus (control) or p16INK4a expressing virus (p16) and selected for 1 wk with puromycin. After selection, cells were grown 3 more days without puromycin, and then, cell growth (thymidine uptake) was examined in the presence of different doses of IL-3. Expression of p16INK4a is shown (inset). One result representative of three independent experiments is shown. Effect of bleomycin treatment on body weight in wild-type and CD69-deficient mice. Time course of changes in body weight after bleomycin (BLM) treatment in wild-type (WT) (n = 8) and cluster of differentiation 69 (CD69)-deficient (CD69-/-) mice (n = 6). Results are expressed as the mean (SEM), *P < 0.05. CCR5−/− CD4+CD25+ regulatory T cells fail to promote parasite persistence. RAG2−/− mice were infected in one ear with 103 L. major promastigotes. At the time of infection, WT CD4+CD25− T cells (3 × 105/mouse) were adoptively transferred together with CD4+CD25+ T cells (3 × 104/mouse) obtained from either WT (•) or CCR5−/− mice (○). (A) Lesion sizes were measured at various time points after infection. Results are shown as the mean ± SD of the induration (four to six mice per time point). (B) Parasite numbers were assessed at 7 wk after infection. Numbers represent the mean parasite number per ear (four mice per group). Results represent the mean ± SD. **, P < 0.01, difference from WT mice. Similar results were obtained in three separate experiments performed.
View of the H—Br contacts in [Eu(H2O)6Br(2)2]Br(1), left: four hydrogen bonds link Br1 to water molecules, right: six hydrogen bonds link Br2 to water molecules. All displacement ellipsoids are drawn at the 90% propability level. [Symmetry codes: (i) -x, y, 1/2 - z; (ii) -x, -y, -z; (iii) x, -y, 1/2 + z.] Mutants possessing a higher aggregation propensity correlate with shorter disease duration. (A) Mutations with a significant number of patients (more than five) grouped in terms or aggregation propensity categories, as explained in Figure 4. Mutations associated with shorter disease durations belong to a group that present high or extreme aggregation propensities. (B) Non-linear regression of aggregation propensity and disease duration. A statistically significant correlation between aggregation and disease duration was not found (P > 0.05). Effect of temperature on activity and stability of purified chitinase from A. faecalis AU02. Values are mean ± SD, n = 3 Kinetics of TREM-1 expression after TLR agonist stimulation in vitro. Displacement of stalled transcription elongation complexes by MfdΔD7 in vitro. Transcription complexes stalled at +20 were formed on an end-labelled fragment of pSRTB1, 250 nM Mfd or MfdΔD7 was added and aliquots were removed and analysed by EMSA. Data points represent the percentage of elongation complex at each time point relative to the amount at t = 0, and are expressed as an average of at least two experiments (shown with data range). The graph shows displacement of wild-type RNAP by MfdΔD7 (filled diamonds) and RNAP βIA117 KA118 EA119 (mt RNAP) by MfdΔD7 (filled triangles) and Mfd (open squares).
Constant PMH error (deg) with standard error as a function of Trial for 135° conditions. Effect of physiological elevation (48 h) in the plasma FFA concentration (brought about by lipid infusion) on plasma C-peptide concentration (left) and insulin secretory response (deconvolution of the palsma C-peptide curve) (right) in offspring of two type 2 diabetic parents (24). Schematic representation of epigenetic mechanisms. (A) In the nucleus, DNA coils and condenses around histones. Each octameric histone core contains two copies each of histones H2A, H2B, H3, and H4. The DNA–protein complex is referred to as chromatin. (B) The DNA-histone interaction occurs at the N-terminal tail of a histone, where for example on the H3 N-terminal tail, there are several sites for epigenetic marking via acetylation, methylation, and phosphorylation. (C) In and around gene promoters that are rich in cytosine-guanine nucleotides (CpG islands), methyl groups are transferred to CpG sites. This process, called DNA methylation, is catalyzed by a class of enzymes known at DNA methyltransferases. Flow diagram of treatment protocol. Performance on the accelerating Rotarod was unaffected by SNCA-overexpression or by chronic NAC treatment.Wild-type (WT) and SNCA-overexpressing (TG) mice treated with either Alanine (ALA) or NAC were tested at 1 year of age on a Rotarod (Ugo Basile) accelerating from 2–40 rpm over a period of 240 seconds. There were no significant differences between the groups tested. WT ALA N = 13, WT NAC N = 14, TG ALA N = 12, TG NAC N = 16.
Conversion of tethering events to rolling is a function of wall shear stress and site density of PNAd. Tethers were considered to result in rolling when neutrophils moved for at least 2 s at a mean velocity of at least fivefold lower than the mean hydrodynamic velocity of untethered cells at a given shear stress. Fat topography in type 2 diabetes and effect of thiazolidine-diones (TZD) (see text for a detailed discussion) The Tg restores splenic B cells in JHD mice. Spleen cells from JHD (A), mIgM (B), and MRL/lpr (C) mice were analyzed via flow cytometry. Live (propidium iodide–negative) cells were analyzed for CD19 (PE) and B220 (FITC) expression. B cells are B220+/CD19+ (upper right quadrant). Percentages of B cells among live splenocytes are shown. In MRL/lpr mice, B220+/CD19− cells are T cells. Wagner-Meerwein skeletal rearrangement of methyl 4-oxo-10-(2-oxopyrrolidinyl)-10H-2,11b-epoxyoxireno[6,7]isoindolo[2,1-a]quinoline-3-carboxylate. The molecular structure of the title compound, showing the atomic numbering scheme. Displacement ellipsoids are drawn at the 50% probability level.
Lister Hill National Center for Biomedical Communications
U.S. National Library of Medicine, 8600 Rockville Pike, Bethesda, MD 20894
National Institutes of Health, Department of Health & Human Services
Privacy, Accessibility, Frequently Asked Questions, Contact Us, Collection
Freedom of Information Act, USA.gov